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This is because the combustion of fossil fuels resulted in increased atmospheric concentrations of carbon dioxide. The Kyoto Protocol was negotiated with the intention that it would reduce the concentration of atmospheric carbon dioxide to below 500 ppm by the year 2010. However, current estimates of future global oil supply make it clear that by 2010 the maximum concentration of atmospheric carbon dioxide that can be achieved with current methods of producing oil is not within this goal. In fact, the time for atmospheric carbon dioxide to reach the Kyoto goal of below 500 ppm is now rapidly passing. Government and the oil and gas industry alike are turning their attention to the possibility of achieving the concentration of atmospheric carbon dioxide to below 350 ppm by the year 2050. In order to achieve this global goal, new and improved technologies for the in situ conversion of fossil carbon into other useful products are urgently needed. Current methods of improving oil recovery may achieve a very small amount of energy from the fuel that is used, but cannot reduce the atmospheric concentration of carbon dioxide. Other methods have been devised that use carbon monoxide or hydrogen to catalytically convert carbon dioxide into gases that can be sold at the well site or the crude plant. One such method involves the use of iron to convert carbon monoxide to carbon dioxide and hydrogen in the presence of a metal oxide catalyst. This invention meets the need for a low cost, new technology for the in situ conversion of fossil fuels into other products by providing a fluid-bed reactor for the catalytic conversion of carbon dioxide, carbon monoxide and hydrogen into graphite, methanol, formaldehyde and water.Functional Roles of miRNAs in Alzheimer's Disease. Alzheimer's disease (AD) is a neurodegenerative disorder that affects multiple brain regions. Genetic and environmental factors contribute to the onset and progression of AD. Numerous studies have provided convincing evidence of the pathophysiology of AD, including the proteinopathies and the cellular mechanisms underlying amyloid beta peptide (Aβ) production 648931e174
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